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 Post subject: Heart Disease theory
PostPosted: Wed Sep 19, 2018 7:22 pm 
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Dr. Levy along with his predecessor Linus Pauling has the best unifying theory on cholesterol and heart disease, I have ever read.

They say that coronary heart disease is caused by a localized scurvy in the damaged blood vessels. Vitamin C is essential to the health of all blood vessels. When the intracellular "glue" of your arteries become damaged due to lack of Vit C, many times from an infection, the first step is arteriosclerosis. Vitamin C deficiency causes an ungelling of the glycoproteins in the basement membrane of the blood vessels. A focal vitamin C deficiency in the arterial wall degenerates the basement membrane allowing the abnormal deposition of solutes such as calcium, cholesterol, and fats because these are substitutes to Vit C. Subsequently there is a proliferation of macrophages in the basement membrane which continues as long the abnormal deposition of these solutes continues. And, until vitamin C levels in the arteries normalize, abnormal deposits will continue to appear and macrophages will continue to proliferate and engulf the solutes (cholesterol), there by continuing the relentless progression of arteriosclerosis.

Cholesterol is just one of a number of substances that will readily deposit n an area of the artery where atherosclerosis has begun. Lipoprotein (a) seems to serve as a surrogate or substitute for vitamin C. Low Vit C levels regularly resulted in a compensatory increase in Lp(a) plasma levels. The body compensates for a lack of Vit C by pulling Lp(a) out of the plasma and depositing it in the wall.


ps: Antifungal means anticholesterol! The anti-cholesterol drugs were discovered accidentally because researchers were looking for antifungal medications! They found the statin group work against fungi because they inhibit an enzyme that our bodies need to process cholesterol. Fungi depend on cholesterol for survival Stopping cholesterol production in the fungi is an effective antifungal mechanism. Statin drugs are fungally derived and are mycotoxins. Fungi can produce cholesterol as well., So the ability of statin drugs to inhibit the enzyme and kill fungi at the same time may be the complex mechanism for statins,,,,,,,not simply a lowering of cholesterol. In short, Kauffman suggests that high cholesterol in the human may also be fungi related. A fungal infection involved in this complex syndrome?

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